The long noncoding RNA TUG1 acts as a competing endogenous RNA to regulate the Hedgehog pathway by targeting miR-132 in hepatocellular carcinoma

نویسندگان

  • Jingjing Li
  • Qinghui Zhang
  • Xiaoming Fan
  • Wenhui Mo
  • Weiqi Dai
  • Jiao Feng
  • Liwei Wu
  • Tong Liu
  • Sainan Li
  • Shizan Xu
  • Wenwen Wang
  • Xiya Lu
  • Qiang Yu
  • Kan Chen
  • Yujing Xia
  • Jie Lu
  • Yingqun Zhou
  • Ling Xu
  • Chuanyong Guo
چکیده

Emerging evidence shows that the Hedgehog pathway and the long noncoding RNA TUG1 play pivotal roles in cell proliferation, migration, and invasion in tumors. However, the mechanism underlying the effect of TUG1 and the Hedgehog pathway in hepatoma remains undefined. In the present study, we showed that the expression of TUG1 was negatively correlated with that of microRNA (miR)-132, and depletion of TUG1 inhibited the activation of the Hedgehog pathway in vitro and in vivo. We showed that TUG1 functions as a competing endogenous (ceRNA) by competing with miR-132 for binding to the sonic hedgehog protein in HCC, thereby suppressing the activation of Hedgehog signaling and its tumorigenic effect. These data indicate that targeting the TUG1-miR132-Hedgehog network could be a new strategy for the treatment of HCC.

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عنوان ژورنال:

دوره 8  شماره 

صفحات  -

تاریخ انتشار 2017